TY - JOUR
T1 - Intraspinal dissemination of intracranial hemangiopericytoma
T2 - Case report and literature review
AU - Ali, Hosam Shata Mohanmed
AU - Endo, Toshiki
AU - Endo, Hidenori
AU - Murakami, Kensuke
AU - Tominaga, Teiji
N1 - Publisher Copyright:
©2016 Surgical Neurology International .
PY - 2016
Y1 - 2016
N2 - Background: The authors report the case of a 53-year-old woman suffering from thoracic myelopathy caused by intraspinal dissemination of hemangiopericytoma. In literature, hemangiopericytoma is commonly found as an intracranial lesion, and often hematogenously metastasizes to the bone or liver; however, intradural spinal dissemination is extremely rare. Case Description: The patient presented with gait disturbance due to thoracic myelopathy 6 years after surgical treatment for intracranial hemangiopericytoma. Magnetic resonance imaging demonstrated intradural disseminated lesions compressing the spinal cord. Although the patient underwent resection of the intradural spinal tumor, the lesion was tightly adherent to the dorsal surface of the spinal cord. Therefore, it resulted in subtotal removal. Immediately after the surgery, symptoms related to the thoracic myelopathy resolved. The patient was free from disease progression for 14 months after whole spine radiotherapy. Conclusion: Recognition of this type of progression is important in the clinical management of intracranial hemangiopericytoma because intradural spinal dissemination dramatically degrades neurological functions.
AB - Background: The authors report the case of a 53-year-old woman suffering from thoracic myelopathy caused by intraspinal dissemination of hemangiopericytoma. In literature, hemangiopericytoma is commonly found as an intracranial lesion, and often hematogenously metastasizes to the bone or liver; however, intradural spinal dissemination is extremely rare. Case Description: The patient presented with gait disturbance due to thoracic myelopathy 6 years after surgical treatment for intracranial hemangiopericytoma. Magnetic resonance imaging demonstrated intradural disseminated lesions compressing the spinal cord. Although the patient underwent resection of the intradural spinal tumor, the lesion was tightly adherent to the dorsal surface of the spinal cord. Therefore, it resulted in subtotal removal. Immediately after the surgery, symptoms related to the thoracic myelopathy resolved. The patient was free from disease progression for 14 months after whole spine radiotherapy. Conclusion: Recognition of this type of progression is important in the clinical management of intracranial hemangiopericytoma because intradural spinal dissemination dramatically degrades neurological functions.
KW - Hemangiopericytoma
KW - myelopathy
KW - spinal dissemination
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U2 - 10.4103/2152-7806.195585
DO - 10.4103/2152-7806.195585
M3 - Article
AN - SCOPUS:85009114634
VL - 7
SP - S1016-S1020
JO - Surgical Neurology International
JF - Surgical Neurology International
SN - 2152-7806
IS - 41
ER -