Progressing small vessel pontine infarction includes different etiologies

Taizen Nakase; Masahiro Sasaki; Yasuko Ikeda; Akifumi Suzuki

doi:10.1002/acn3.25

Progressing small vessel pontine infarction includes different etiologies

Taizen Nakase, Masahiro Sasaki, Yasuko Ikeda, Akifumi Suzuki

Research output: Contribution to journal › Article › peer-review

Abstract

Background: The aim of this study was to investigate the clinical features of progressing stroke of pontine infarction as small vessel disease. Methods: Enrolled 38 acute pontine infarctions were confirmed by magnetic resonance imaging and magnetic resonance angiography at the first and seventh days. Ten patients (26.3%) presented progression (NIH Stroke Scale ≥2 increase within 72 h). Results: Progressing patients showed no relation to the size and the distribution of lesion. Expansion of ischemic lesion showed correlation with basilar artery atherosclerosis. Stable lesion related to delayed worsening. Conclusion: These findings suggest that progressing stroke may be caused by not only the worsening of blood flow of ischemic lesion but also delayed neuronal death.

Original language	English
Pages (from-to)	75-79
Number of pages	5
Journal	Annals of Clinical and Translational Neurology
Volume	1
Issue number	2
DOIs	https://doi.org/10.1002/acn3.25
Publication status	Published - 2014 Feb
Externally published	Yes

ASJC Scopus subject areas

Neuroscience(all)
Clinical Neurology

Access to Document

10.1002/acn3.25

Fingerprint Dive into the research topics of 'Progressing small vessel pontine infarction includes different etiologies'. Together they form a unique fingerprint.

Cite this

@article{aeb0f79cf596490c988e2fd56263b183,

title = "Progressing small vessel pontine infarction includes different etiologies",

abstract = "Background: The aim of this study was to investigate the clinical features of progressing stroke of pontine infarction as small vessel disease. Methods: Enrolled 38 acute pontine infarctions were confirmed by magnetic resonance imaging and magnetic resonance angiography at the first and seventh days. Ten patients (26.3%) presented progression (NIH Stroke Scale ≥2 increase within 72 h). Results: Progressing patients showed no relation to the size and the distribution of lesion. Expansion of ischemic lesion showed correlation with basilar artery atherosclerosis. Stable lesion related to delayed worsening. Conclusion: These findings suggest that progressing stroke may be caused by not only the worsening of blood flow of ischemic lesion but also delayed neuronal death.",

author = "Taizen Nakase and Masahiro Sasaki and Yasuko Ikeda and Akifumi Suzuki",

note = "Publisher Copyright: {\textcopyright} 2013 The Authors.",

year = "2014",

month = feb,

doi = "10.1002/acn3.25",

language = "English",

volume = "1",

pages = "75--79",

journal = "Annals of Clinical and Translational Neurology",

issn = "2328-9503",

publisher = "John Wiley and Sons Ltd",

number = "2",

}

TY - JOUR

T1 - Progressing small vessel pontine infarction includes different etiologies

AU - Nakase, Taizen

AU - Sasaki, Masahiro

AU - Ikeda, Yasuko

AU - Suzuki, Akifumi

PY - 2014/2

Y1 - 2014/2

N2 - Background: The aim of this study was to investigate the clinical features of progressing stroke of pontine infarction as small vessel disease. Methods: Enrolled 38 acute pontine infarctions were confirmed by magnetic resonance imaging and magnetic resonance angiography at the first and seventh days. Ten patients (26.3%) presented progression (NIH Stroke Scale ≥2 increase within 72 h). Results: Progressing patients showed no relation to the size and the distribution of lesion. Expansion of ischemic lesion showed correlation with basilar artery atherosclerosis. Stable lesion related to delayed worsening. Conclusion: These findings suggest that progressing stroke may be caused by not only the worsening of blood flow of ischemic lesion but also delayed neuronal death.

AB - Background: The aim of this study was to investigate the clinical features of progressing stroke of pontine infarction as small vessel disease. Methods: Enrolled 38 acute pontine infarctions were confirmed by magnetic resonance imaging and magnetic resonance angiography at the first and seventh days. Ten patients (26.3%) presented progression (NIH Stroke Scale ≥2 increase within 72 h). Results: Progressing patients showed no relation to the size and the distribution of lesion. Expansion of ischemic lesion showed correlation with basilar artery atherosclerosis. Stable lesion related to delayed worsening. Conclusion: These findings suggest that progressing stroke may be caused by not only the worsening of blood flow of ischemic lesion but also delayed neuronal death.

UR - http://www.scopus.com/inward/record.url?scp=84978032099&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=84978032099&partnerID=8YFLogxK

U2 - 10.1002/acn3.25

DO - 10.1002/acn3.25

M3 - Article

AN - SCOPUS:84978032099

VL - 1

SP - 75

EP - 79

JO - Annals of Clinical and Translational Neurology

JF - Annals of Clinical and Translational Neurology

SN - 2328-9503

IS - 2

ER -

Progressing small vessel pontine infarction includes different etiologies

Abstract

ASJC Scopus subject areas

Access to Document

Other files and links

Cite this