Insulin signaling: GLUT4 vesicles exit via the exocyst

Makoto Kanzaki, Jeffrey E. Pessin

Research output: Contribution to journalReview articlepeer-review

50 Citations (Scopus)

Abstract

The localization of the GTP-binding protein TC10 to lipid raft microdomains has been suggested to play a role in the stimulation of GLUT4 translocation. The exocyst has now been identified as a downstream target for TC10, directing GLUT4-containing vesicles to the site of fusion.

Original languageEnglish
Pages (from-to)R574-R576
JournalCurrent Biology
Volume13
Issue number14
DOIs
Publication statusPublished - 2003 Jul 15
Externally publishedYes

ASJC Scopus subject areas

  • Biochemistry, Genetics and Molecular Biology(all)
  • Agricultural and Biological Sciences(all)

Fingerprint Dive into the research topics of 'Insulin signaling: GLUT4 vesicles exit via the exocyst'. Together they form a unique fingerprint.

Cite this