Influence of total biliary diversion on the pancreatic exocrine secretion and pancreatic growth in dogs

M. Takahashi; I. Sasaki; H. Naito; C. Shibata; T. Doi; S. Matsuno

Influence of total biliary diversion on the pancreatic exocrine secretion and pancreatic growth in dogs

M. Takahashi, I. Sasaki, H. Naito, C. Shibata, T. Doi, S. Matsuno

MED - Medical Sciences

Research output: Contribution to journal › Article › peer-review

Abstract

Chronic Total Biliary Diversion induced both pancreatic trophic effect and exocrine pancreatic hypersecretion coincided with augmented plasma CCK levels. Intravenous CCK receptor antagonist (CR 1505) administration suppressed Na oleate stimulated exocrine pancreatic secretion in TBD dogs. We conclude that intraluminal bile will regulate exocrine pancreatic secretion and may affect pancreatic growth by regulating CCK release in dogs.

Original language	English
Pages (from-to)	391-396
Number of pages	6
Journal	Biomedical Research
Volume	16
Issue number	SUPPL. 2
Publication status	Published - 1995 Jan 1

ASJC Scopus subject areas

Biochemistry, Genetics and Molecular Biology(all)

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title = "Influence of total biliary diversion on the pancreatic exocrine secretion and pancreatic growth in dogs",

abstract = "Chronic Total Biliary Diversion induced both pancreatic trophic effect and exocrine pancreatic hypersecretion coincided with augmented plasma CCK levels. Intravenous CCK receptor antagonist (CR 1505) administration suppressed Na oleate stimulated exocrine pancreatic secretion in TBD dogs. We conclude that intraluminal bile will regulate exocrine pancreatic secretion and may affect pancreatic growth by regulating CCK release in dogs.",

author = "M. Takahashi and I. Sasaki and H. Naito and C. Shibata and T. Doi and S. Matsuno",

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AU - Shibata, C.

AU - Doi, T.

AU - Matsuno, S.

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N2 - Chronic Total Biliary Diversion induced both pancreatic trophic effect and exocrine pancreatic hypersecretion coincided with augmented plasma CCK levels. Intravenous CCK receptor antagonist (CR 1505) administration suppressed Na oleate stimulated exocrine pancreatic secretion in TBD dogs. We conclude that intraluminal bile will regulate exocrine pancreatic secretion and may affect pancreatic growth by regulating CCK release in dogs.

AB - Chronic Total Biliary Diversion induced both pancreatic trophic effect and exocrine pancreatic hypersecretion coincided with augmented plasma CCK levels. Intravenous CCK receptor antagonist (CR 1505) administration suppressed Na oleate stimulated exocrine pancreatic secretion in TBD dogs. We conclude that intraluminal bile will regulate exocrine pancreatic secretion and may affect pancreatic growth by regulating CCK release in dogs.

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