Correction to: the IL1RN Mutation Creating the Most-Upstream Premature Stop Codon Is Hypomorphic because of a Reinitiation of Translation (Journal of Clinical Immunology, (2020), 40, 4, (643-645), 10.1007/s10875-020-00770-1)

Kunihiko Moriya, Saori Kadowaki, Tomohiro Nakano, Sanem E. Akarcan, Necil Kutukculer, Guzide Aksu, Yoji Sasahara, Shigeo Kure, Hidenori Ohnishi, Jean Laurent Casanova, Anne Puel, Toshiyuki Fukao

Research output: Contribution to journalComment/debatepeer-review

Abstract

The original version of our manuscript, entitled, “ The IL1RN mutation creating the most-upstream premature stop codon is hypomorphic because of a reinitiation of translation” unfortunately contained mistakes in Fig. 1a and d legends. The text should read as follows: a. Immunoblot analysis with an anti-FLAG antibody of HEK- 293 T cells transfected with insert-free FLAG-tagged vector (mock) or with expression vectors carrying various IL1RN alleles:WT, R29X, or R29X-M38A (methionine 38 to alanine mutation, blocking reinitiation of translation at M38) or E57X. d. Protein structure model of IL-1Ra and IL-1R1. IL-1Ra (blue) was superimposed on EBI-005 in complex with IL-1R1 (green).

Original languageEnglish
Pages (from-to)646
Number of pages1
JournalJournal of Clinical Immunology
Volume40
Issue number4
DOIs
Publication statusPublished - 2020 May 1

ASJC Scopus subject areas

  • Immunology and Allergy
  • Immunology

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