A novel cell death gene acts to repair patterning defects in Drosophila melanogaster

Kentaro M. Tanaka; Aya Takahashi; Naoyuki Fuse; Toshiyuki Takano-Shimizu-Kouno

doi:10.1534/genetics.114.163337

A novel cell death gene acts to repair patterning defects in Drosophila melanogaster

Kentaro M. Tanaka, Aya Takahashi, Naoyuki Fuse, Toshiyuki Takano-Shimizu-Kouno

Research output: Contribution to journal › Comment/debate › peer-review

4 Citations (Scopus)

Abstract

Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.

Original language	English
Pages (from-to)	739-742
Number of pages	4
Journal	Genetics
Volume	197
Issue number	2
DOIs	https://doi.org/10.1534/genetics.114.163337
Publication status	Published - 2014 Jun
Externally published	Yes

ASJC Scopus subject areas

Genetics

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title = "A novel cell death gene acts to repair patterning defects in Drosophila melanogaster",

abstract = "Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.",

author = "Tanaka, {Kentaro M.} and Aya Takahashi and Naoyuki Fuse and Toshiyuki Takano-Shimizu-Kouno",

year = "2014",

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AU - Tanaka, Kentaro M.

AU - Takahashi, Aya

AU - Fuse, Naoyuki

AU - Takano-Shimizu-Kouno, Toshiyuki

PY - 2014/6

Y1 - 2014/6

N2 - Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.

AB - Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.

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DO - 10.1534/genetics.114.163337

M3 - Comment/debate

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JO - Genetics

JF - Genetics

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ER -

A novel cell death gene acts to repair patterning defects in Drosophila melanogaster

Abstract

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