A novel cell death gene acts to repair patterning defects in Drosophila melanogaster

Kentaro M. Tanaka, Aya Takahashi, Naoyuki Fuse, Toshiyuki Takano-Shimizu-Kouno

Research output: Contribution to journalComment/debate

1 Citation (Scopus)

Abstract

Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.

Original languageEnglish
Pages (from-to)739-742
Number of pages4
JournalGenetics
Volume197
Issue number2
DOIs
Publication statusPublished - 2014 Jun
Externally publishedYes

ASJC Scopus subject areas

  • Genetics

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