Abstract
Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.
Original language | English |
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Pages (from-to) | 739-742 |
Number of pages | 4 |
Journal | Genetics |
Volume | 197 |
Issue number | 2 |
DOIs |
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Publication status | Published - 2014 Jun |
Externally published | Yes |
ASJC Scopus subject areas
- Genetics